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Research Points to Potential Therapy for Tumor-Associated Epilepsy


ScienceDaily (Sep. 12, 2011) — Glioma, one of the most deadly and common types of brain tumor, is often associated with seizures, but the origins of these seizures and effective treatments for them have been elusive. Now a team funded by the National Institutes of Health has found that human gliomas implanted in mice release excess levels of the brain chemical glutamate, overstimulating neurons near the tumor and triggering seizures.[U1]–The researchers also found that sulfasalazine, a drug on the market for treating certain inflammatory disorders, can reduce seizures in mice with glioma.—About 80 percent of people with glioma will experience at least one seizure during their illness, often as the first symptom. About one-third of patients will develop recurring seizures, known as tumor-associated epilepsy. Sen. Ted Kennedy, D-Mass., whose death was caused by a malignant glioma in August 2009, was diagnosed after having a seizure 15 months earlier.—“Seizures are a frequent symptom of glioma and are often poorly controlled by epilepsy medications,” said Jane Fountain, Ph.D., a program director at NIH’s National Institute of Neurological Disorders and Stroke (NINDS). “Understanding why the seizures occur and how to counteract them could help us substantially improve the quality of life for people with glioma.” “People have assumed that tumors cause seizures by irritating the brain, but that really isn’t a scientific explanation. We have now shown that the seizures are caused by glutamate release from the tumor,” said Harald Sontheimer, Ph.D., a professor of neurobiology and director of the Center for Glial Biology in Medicine at the University of Alabama Birmingham (UAB). Dr. Sontheimer and his team published their results in Nature Medicine.—The research was supported by NINDS, including $934,698 in grants funded through the American Recovery and Reinvestment Act.—Glutamate serves as a chemical relay within the brain. Its release from one neuron can stimulate other neurons to fire electrical impulses. However, excess glutamate can cause abnormal electrical activity in the brain — which is the basis for epileptic seizures. In particular, excess release of glutamate from non-neuronal cells called glia appears to play a role in some types of epilepsy. Because gliomas result from an overgrowth of glia, researchers had theorized that glutamate produced by the tumors might cause seizures, but no one had established a causal link.—Dr. Sontheimer’s team tested the theory by studying mice whose brains were seeded with human glioma cells. They found that about one-third of the animals with gliomas developed abnormal brain activity and behavioral signs consistent with seizures. They also investigated whether or not the tumors affect brain activity in response to stimulation. When they delivered electrical pulses near a tumor, they saw a pattern of activity that spread outward from the tumor and was more prolonged and widespread than the responses to stimulation seen in normal brain tissue. Brain tissue containing the tumors also released higher levels of glutamate compared to normal brain tissue.—Next, the researchers sought to determine if the drug sulfasalazine could correct these abnormalities. Sulfasalazine is an anti-inflammatory sometimes prescribed for ulcerative colitis and rheumatoid arthritis. It also targets a protein complex called the system Xc(-) transporter. System Xc(-) acts like a -commodities broker within glioma cells, importing the essential amino acid cystine into the cells in exchange for exporting glutamate.[U2]–Dr. Sontheimer’s team found that by inhibiting the system Xc(-) transporter, sulfasalazine can reduce glutamate release from gliomas. The drug also reduced seizure activity in the glioma-bearing mice, cutting the frequency of epileptic bursts nearly fivefold in the first hour after treatment. After four hours, the effects of the drug wore off unless it was re-administered. The likely reason is that most of the drug is broken down into a form that does not affect system Xc(-), according to Dr. Sontheimer. A clinical trial is planned at UAB to determine if sulfasalazine can reduce seizures in people with slow-growing gliomas. Meanwhile, Dr. Sontheimer’s lab is working with medicinal chemists to develop a form of the drug that is more stable in the bloodstream and brain, and more active against system Xc(-).—“There is hope that in addition to reducing seizures, sulfasalazine might reduce the growth of glioma cells,” Dr. Sontheimer said. The cystine molecules imported by system Xc(-) are used to manufacture vital proteins that help tumor cells grow stronger, he explained. In a 2005 study, he found that sulfasalazine delays glioma growth in mice.—Whether these promising results with sulfasalazine in animal studies will translate into improved outcome in patients with brain tumors remains to be tested. Indeed, caution was raised by a small trial of 10 patients with advanced stage gliomas treated with varying doses of sulfasalazine. No beneficial effects were established, and safety concerns arose about whether treatment worsened brain swelling near the tumor. That trial was terminated early.—“It is worth examining whether or not the drug can help patients with newly diagnosed, slow-growing gliomas as opposed to patients with advanced disease,” Dr. Sontheimer said.—The lead author of the study, Susan Buckingham, Ph.D., and second author, Susan Campbell, Ph.D., received support from the NINDS Training Program in Brain Tumor Biology at UAB. Vedrana Montana, Ph.D., was hired as a postdoctoral fellow in the Center for Glial Biology in Medicine through Recovery Act funds. The work was also supported by a center core grant from the NIH Blueprint for Neuroscience Research.–
















And an interesting concept as well about the temple of our bodies are belonging go God and we are his temple and not to defile it

Interesting perspectives on the vaccines in any form should then be not initiated

If you believe!!



Chemists Help Astronauts Make Sure Their Drinking Water Is Clean


Nicole Stott, a flight engineer on Expedition 21 of the International Space Station, tests the quality of drinking water using chemistry and procedures developed by Iowa State University and Ames Laboratory researchers.—ScienceDaily (Sep. 15, 2011) — Bob Lipert held up a syringe, attached a plastic cartridge and demonstrated how chemistry developed at Iowa State University is helping astronauts and cosmonauts make sure they have safe drinking water at the International Space Station.—Each cartridge contains a thin, one-centimeter disk that’s loaded with chemistry, said Lipert, an associate scientist with Iowa State’s Institute for Physical Research and Technology and an associate of the U.S. Department of Energy’s Ames Laboratory. Run a 10-milliliter water sample through a disk and it will change color in the presence of iodine, which NASA uses to inhibit the growth of microorganisms in the drinking water stored at the space station. The disk will turn from white to yellow and, as it’s exposed to higher concentrations of iodine, it will turn to orange and finally to a rust color.—A handheld device — a diffuse reflectance spectrometer — can read the disk’s color changes and precisely measure the concentration of molecular iodine, or I2. The whole process is called colorimetric solid phase extraction.—Starting in late September, Lipert said astronauts at the space station will use new developments and procedures that convert all forms of iodine in the water samples to molecular iodine. That will give astronauts a more precise reading of total iodine in their drinking water. Lipert said they’ll know in real time whether there’s too much, too little or just enough iodine in the water.—Disks loaded with different chemistry can also measure and record concentrations of silver, which the Russian Federal Space Agency uses as a biocide in its water supply at the space station. As silver concentrations increase, disks turn from yellow to purple.

Before Iowa State chemists helped develop the new tests, the only way to test the space station’s drinking water was to send samples back to earth.—“We figured out the chemistry and put it into a form that can be used in space,” Lipert said. “We also took lab techniques and simplified them as much as possible. And we developed procedures that can be used in the absence of gravity.”—The result is a quick, accurate test that doesn’t use up much drinking water or much astronaut time.—“What’s neat about what we came up with is that all the chemistry we need to do can be accomplished in about one minute per sample using a little, 1-centimeter cartridge,” Lipert said.—It took some work to develop the test’s chemistry and procedures. The NASA-sponsored project began more than a decade ago under the direction of Marc Porter, a former Iowa State professor of chemistry and chemical and biological engineering who is now a USTAR Professor at the University of Utah in Salt Lake City. Lipert has worked on the project since 2000.—Other Iowa State researchers who have worked on the project include Jim Fritz, Distinguished Professor Emeritus of Liberal Arts and Sciences; former post-doctoral researchers Matteo Arena and Neil Dias; and former graduate students April Hill, Daniel Gazda, John Nordling, Lisa Ponton and Cherry Shih. Lorraine Siperko, a research scientist at the University of Utah, has also worked on the project.—The university researchers have also collaborated with the Wyle Integrated Science and Engineering Group, a NASA subcontractor that helped develop and certify the water-testing hardware that has been deployed on the space station.—After a series of successful space tests in 2009 and ’10, the researchers’ water-testing equipment is now certified operational hardware and is part of the space station’s environmental monitoring toolbox.—Lipert said the testing technology can also be a useful tool in many earthbound applications, including forensics tests for drugs, environmental tests for heavy metals and water quality tests for pesticides or herbicides.—“This is a very flexible platform,” he said. “You just have to work out the chemistry for each substance you’re analyzing.”Story Source:

The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Iowa State University.




When a Salad Is Not a Salad: Why Are Dieters Easily Misled by Food Names?


ScienceDaily (Apr. 29, 2011) — Dieters are so involved with trying to eat virtuously that they are more likely than non-dieters to choose unhealthy foods that are labeled as healthy, according to a new study in the Journal of Consumer Research. It seems dieter focus on food names can work to their disadvantage.—“Keeping your weight-loss goal in mind as you scan the lunch menu at a café, you are careful to avoid pasta selections and instead order from the list of salad options,” write authors Caglar Irmak (University of South Carolina), Beth Vallen (Loyola University), and Stefanie Rosen Robinson (University of South Carolina). “But before you congratulate yourself for making a virtuous selection, you might want to consider whether your choice is a salad in name only.”–These days, restaurant salads can include ingredients that dieters would be likely to avoid (meats, cheeses, breads, and pasta). Potato chips are labeled “veggie chips,” milkshakes are called “smoothies,” and sugary drinks are named “flavored water.” Why are dieters, who are supposedly more attuned to healthy foods, likely to be confused by these labels?—“Over time, dieters learn to focus on simply avoiding foods that they recognize as forbidden based on product name,” the authors explain. “Thus, dieters likely assume that an item assigned an unhealthy name (for example, pasta) is less healthy than an item assigned a healthy name (for example, salad), and they do not spend time considering other product information that might impact their product evaluations.” Non-dieters do not learn to avoid foods based on names and, given that they are not focused on healthful eating, are more likely to dismiss cues that imply healthfulness, including name.—Participants in one study were presented with a mixture of vegetables, pasta, salami, and cheese, served on a bed of fresh romaine lettuce. The item was either identified as “salad” or “pasta.” When it was called pasta, dieters perceived it as less healthy. In another study, participants were given samples of a product, which was labeled either “fruit chews” or “candy chews.” “Dieters perceived the item with an unhealthy name (candy chews) to be less healthful and less tasty than non-dieters,” the authors write. As a result, dieters consumed more of the confections when they were called “fruit chews.”–Journal ReferenceCaglar Irmak, Beth Vallen, and Stefanie Rosen Robinson. The Impact of Product Name on Dieters’ and Non-Dieters’ Food Evaluations and Consumption. Journal of Consumer Research, October 2011 DOI: 10.1086/660044



Recipe—How to purify Water with Iodine or Chorine or Colloidal Silver or Food Grade Hydrogen Peroxide


Calculate how much iodine is needed to purify the water. Consider the source of the water and the amount of water. When using 2 percent liquid iodine, five drops from an eyedropper should be added for each quart if the water is clear and from a flowing source. If the water is cloudy or from a stagnant source, use 10 drops. If using tablets, use two tablets per quart.–Let the water sit for 30 minutes, longer if the water was less than 68 degrees Fahrenheit. As temperature drops below 68 degrees Fahrenheit, iodine purification becomes less effective. Water can be warmed by letting it sit in the sun. or heating it –Shake the water and iodine solution thoroughly.—Let solution stand an additional 30 minutes if the water used was especially cloudy, or if it came from a highly contaminated source. The Center for Disease Control states that iodine can be just as effective as chlorine if allowed to sit long enough.



Iodine Solution – One of the most common methods, Using a 2% solution use 5 drops per litre of water. Leave to stand for 15 minutes before drinking. If the water is very cold or cloudy then leave it for 30 minutes, or alternatively use double the amount of drops.




Most household chlorine bleaches have 4-6 percent available chlorine in which case add 1/8 teaspoon (8 drops) of regular, unscented, liquid household bleach for each gallon of water (2 drops per litre or quart if in the USA), stir it well and let it stand for 30 minutes before you use it. Check the label; if the percentage of available chlorine is around 1 percent, or you don’t know what the percentage is, use 40 drops per gallon/ 10 drops per litre; if the percentage is 7-10 percent, use 4 drops per gallon or 1 drop per litre. Double the amount of chlorine if the water is cloudy, murky, or colored, or if the water is extremely cold. If, after sitting covered for 30 minutes, the water doesn’t have a slight chlorine odor, repeat the dosage and let sit for another 15 minutes.



Chlorine As A Purifier-

1) Clear water is a sign of pure water. Always drain long-standing pipes for 30 seconds to one minute before drinking! (Cheap remote motels?)


2) 1 Gallon water is disinfected by 8-16 drops of regular household bleach (visually about 1/4 of a teaspoon) – double that for cloudy water. Shake and let stand 30 minutes. One teaspoon will disinfect 5 gallons. Immediately after treating, water must initially have a slight smell of chlorine. If it does not – repeat the process.


3) Household bleach is relatively harmless. The smell or �waft� of chlorine is not bad: it indicates that water is treated and germ free. Once treated and disinfected, the chlorine smell will go away in a few days.


4) Regularly used water from large tanks may be treated once or twice a month with 1 Oz. bleach per 200 gallons or 5 Oz. bleach per 1000 gallons.


5) Long-standing water in tanks will be disinfected w/ 1 pint household bleach per 1000 gallons. (2500 gal tanks are fine with 3 pints.)


6) Bleach effectively kills bacteria and viruses, stops smells and then breaks down. It’s effective germ killing alkaline property is completely neutralized very quickly. It does not stay chemically active in tanks for more than a few days. Most germs require sunlight to grow. Store water in the dark.


7)If water is relatively clear: but has a noticeable smell of chlorine: it is drinkable, disinfected, and harmless. Humans need 2 quarts per day.



Colloidal Silver


Colloidal Silver is an excellent water purifier. Water stored with one or two tablespoons of Colloidal Silver per 5 litres will be safe and sweet tasting for a very long time.


Water containing germ contaminants (not toxic chemicals) can be made drinkable by adding 2 to 3 tablespoons of Colloidal Silver to every 5 litres of water.



Hydrogen Peroxide


Hydrogen Peroxide method. Food grade hydrogen peroxide is excellent for treating water. Use the same amounts as iodine ( that would be 4 drops to a quart ). Water treated with hydrogen peroxide will last several years longer than any other treatment method.

Recommend 1/8 of a cup of food grade per gallon —Suggested dose at a 32%


How to Concentrate Peroxide

-store bought hydrogen peroxide is usually 3% H2O2, 97% H20

-water freezes at 0 degrees

-H2O2 freezes at -11 degrees


so just stick it in your freezer and when your liquid is frozen just take it out and pour your high concentration H2O2 out. Please remember that at high purity s this can be very dangerous and reactive. Will get about a half an oz with this



21 Water Purification: Chemical Treatment: Hydrogen Peroxide–Hydrogen Peroxide can be used to purify water if nothing else is

available. Studies have shown of 99 percent inactivation of

poliovirus in 6 hr with 0.3 percent hydrogen peroxide and a 99%

inactivation of rhinovirus with a 1.5% solution in 24 minutes.

Hydrogen Peroxide is more effective against bacteria, though Fe+2 or

Cu+2 needs to be present as a catalyst to get a reasonable

concentration-time product.






[U1]Taurine Balances out glutamate
[U2]L Cysteine can be gotten from Garlic being fermented and can be bought at any health food store—here are some sources for this as wll Dairy Products: Most Dairy Products contain Cysteine.

Eggs Yolks

Meats: Most Meats contain Cysteine.

Nuts: Most Nuts contain Cysteine.

Seeds: Most Seeds contain Cysteine.

Vegetables: Garlic Onions





Show of the Week September 26 2011


Caffeine Lowers Risk of Skin Cancer: Coffee-Based Sunscreen Might Work Best


Omega Resources


Different Sources of Omega 3’s


More Mammograms Equal More Mastectomies


Exposure to ‘White’ Light LEDs Appears to Suppress Body’s Production of Melatonin More Than Certain Other Lights


Pituitary Hormone TSH Found to Directly Influence Bone Growth




Caffeine Lowers Risk of Skin Cancer: Coffee-Based Sunscreen Might Work Best


ScienceDaily (Aug. 15, 2011) — There might be a time when instead of just drinking that morning cup of coffee you lather it on your skin as a way of preventing harmful sun damage or skin cancer.—A new Rutgers study strengthens the theory that caffeine guards against certain skin cancers at the molecular level by inhibiting a protein enzyme in the skin, known as ATR. Scientists believe that based on what they have learned studying mice, caffeine applied directly to the skin might help prevent damaging UV light from causing skin cancer.—Prior research indicated that mice that were fed caffeinated water and exposed to lamps that generated UVB radiation that damaged the DNA in their skin cells were able to kill off a greater percentage of their badly damaged cells and reduce the risk of cells becoming cancerous.–“Although it is known that coffee drinking is associated with a decreased risk of non-melanoma skin cancer, there now needs to be studies to determine whether topical caffeine inhibits sunlight-induced skin cancer,” said Allan Conney, director of the Susan Lehman Cullman Laboratory for Cancer Research.—In this newly-published study, instead of inhibiting ATR with caffeinated water, Rutgers researchers, in collaboration with researchers from the University of Washington, genetically modified and diminished ATR in one group of mice. The results: the genetically modified mice developed tumors more slowly than the unmodified mice, had 69 percent fewer tumors than regular mice and developed four times fewer invasive tumors. The study also found, however, that when both groups of mice were exposed to chronic ultraviolet rays for an extended period of time, tumor development occurred in both the genetically modified and regular mice. What this seems to indicate, says Conney, is that inhibiting the ATR enzyme works best at the pre-cancerous stage before UV-induced skin cancers are fully developed.—According to the National Cancer Institute, sunlight-induced skin cancer is the most prevalent cancer in the United States with more than 1 million new cases each year. Although multiple human epidemiologic studies link caffeinated beverage intake with significant decreases in several different types of cancer, including skin cancer, just how and why coffee protects against the disease is unknown. “Caffeine might become a weapon in prevention because it inhibits ATR and also acts ad as a sunscreen and directly absorbs damaging UV light,” said Conney. Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Rutgers University. Journal Reference-Masaoki Kawasumi, Bianca Lemos, James E. Bradner, Renee Thibodeau, Yong-son Kim, Miranda Schmidt, Erin Higgins, Sang-wahn Koo, Aimee Angle-Zahn, Adam Chen, Douglas Levine, Lynh Nguyen, Timothy P. Heffernan, Isabel Longo, Anna Mandinova, Yao-Ping Lu, Allan H. Conney, and Paul Nghiem. Protection from UV-induced skin carcinogenesis by genetic inhibition of the ataxia telangiectasia and Rad3-related (ATR) kinase. Proceedings of the National Academy of Sciences, August 15, 2011 DOI: 10.1073/pnas.1111378108



Omega Resources


Polyunsaturated fatty acids are fatty acids that contain more than one double bond in their backbone. This class includes many important compounds, such as essential fatty acids and those that give drying oils their characteristic property.


Polyunsaturated fatty acids can be classified in various groups by their chemical structure:Contents [hide]

1 Methylene-Interrupted Polyenes

1.1 Omega-3

1.2 Omega-6

1.3 Omega-9

2 Conjugated fatty acids

3 Other Polyunsaturates

4 Function and effects

5 See also

6 References

6.1 Citations

6.2 General References


Methylene-Interrupted Polyenes–These fatty acids have 2 or more cis double bonds that are separated from each other by a single methylene group. (This form is also sometimes called a divinylmethane pattern.) [1] Methylene- interrupted double bonds-C-C=C-C-C=C-The essential fatty acids are all omega-3 and -6 methylene-interrupted fatty acids. See more at Essential fatty acids—




Omega-3 fatty acidsCommon name Lipid name Chemical name

Hexadecatrienoic acid (HTA) 16:3 (n-3) all-cis 7,10,13-hexadecatrienoic acid

Alpha-linolenic acid (ALA) 18:3 (n-3) all-cis-9,12,15-octadecatrienoic acid

Stearidonic acid (SDA) 18:4 (n-3) all-cis-6,9,12,15,-octadecatetraenoic acid

Eicosatrienoic acid (ETE) 20:3 (n-3) all-cis-11,14,17-eicosatrienoic acid

Eicosatetraenoic acid (ETA) 20:4 (n-3) all-cis-8,11,14,17-eicosatetraenoic acid

Eicosapentaenoic acid (EPA, Timnodonic acid) 20:5 (n-3) all-cis-5,8,11,14,17-eicosapentaenoic acid

Heneicosapentaenoic acid (HPA) 21:5 (n-3) all-cis-6,9,12,15,18-heneicosapentaenoic acid

Docosapentaenoic acid (DPA, Clupanodonic acid) 22:5 (n-3) all-cis-7,10,13,16,19-docosapentaenoic acid

Docosahexaenoic acid (DHA, Cervonic acid) 22:6 (n-3) all-cis-4,7,10,13,16,19-docosahexaenoic acid

Tetracosapentaenoic acid 24:5 (n-3) all-cis-9,12,15,18,21-tetracosapentaenoic acid

Tetracosahexaenoic acid (Nisinic acid) 24:6 (n-3) all-cis-6,9,12,15,18,21-tetracosahexaenoic acid




Omega-6 fatty acidsCommon name Lipid name Chemical name

Linoleic acid 18:2 (n-6) all-cis-9,12-octadecadienoic acid

Gamma-linolenic acid (GLA) 18:3 (n-6) all-cis-6,9,12-octadecatrienoic acid

Eicosadienoic acid 20:2 (n-6) all-cis-11,14-eicosadienoic acid

Dihomo-gamma-linolenic acid (DGLA) 20:3 (n-6) all-cis-8,11,14-eicosatrienoic acid

Arachidonic acid (AA) 20:4 (n-6) all-cis-5,8,11,14-eicosatetraenoic acid

Docosadienoic acid 22:2 (n-6) all-cis-13,16-docosadienoic acid

Adrenic acid 22:4 (n-6) all-cis-7,10,13,16-docosatetraenoic acid

Docosapentaenoic acid (Osbond acid) 22:5 (n-6) all-cis-4,7,10,13,16-docosapentaenoic acid

Tetracosatetraenoic acid 24:4 (n-6) all-cis-9,12,15,18-tetracosatetraenoic acid

Tetracosapentaenoic acid 24:5 (n-6) all-cis-6,9,12,15,18-tetracosapentaenoic acid




Omega-9 fatty acids, mono- and polyunsaturatedCommon name Lipid name Chemical name

Oleic acid† 18:1 (n-9) cis-9-octadecenoic acid

Eicosenoic acid† 20:1 (n-9) cis-11-eicosenoic acid

Mead acid 20:3 (n-9) all-cis-5,8,11-eicosatrienoic acid

Erucic acid† 22:1 (n-9) cis-13-docosenoic acid

Nervonic acid† 24:1 (n-9) cis-15-tetracosenoic acid



Conjugated fatty acids

Conjugated double bonds-C=C-C=C-



Conjugated fatty acids have two or more conjugated double bondsCommon name Lipid name Chemical name

Conjugated Linoleic Acids (two conjugated double bonds)

Rumenic acid 18:2 (n-7) 9Z,11E-octadeca-9,11-dienoic acid

18:2 (n-6) 10E,12Z-octadeca-9,11-dienoic acid

Conjugated Linolenic Acids (three conjugated double bonds)

α-Calendic acid 18:3 (n-6) 8E,10E,12Z-octadecatrienoic acid

β-Calendic acid 18:3 (n-6) 8E,10E,12E-octadecatrienoic acid

Jacaric acid 18:3 (n-6) 8E,10Z,12E-octadecatrienoic acid

α-Eleostearic acid 18:3 (n-5) 9Z,11E,13E-octadeca-9,11,13-trienoic acid

β-Eleostearic acid 18:3 (n-5) 9E,11E,13E-octadeca-9,11,13-trienoic acid

Catalpic acid 18:3 (n-5) 9Z,11Z,13E-octadeca-9,11,13-trienoic acid

Punicic acid 18:3 (n-5) 9Z,11E,13Z-octadeca-9,11,13-trienoic acid


Rumelenic acid 18:3 (n-3) 9E,11Z,15E-octadeca-9,11,15-trienoic acid

α-Parinaric acid 18:4 (n-3) 9E,11Z,13Z,15E-octadeca-9,11,13,15-trienoic acid

β-Parinaric acid 18:4 (n-3) all trans-octadeca-9,11,13,15-trienoic acid

Bosseopentaenoic acid 20:5 (n-6) 5Z,8Z,10E,12E,14Z-eicosanoic acid


Other PolyunsaturatesCommon name Lipid name Chemical name

Pinolenic acid 18:3 (n-6) (5Z,9Z,12Z)-octadeca-5,9,12-trienoic acid

Podocarpic acid 20:3 (n-6) (5Z,11Z,14Z)-eicosa-5,11,14-trienoic ac



Different Sources of Omega 3’s


Omega 3 Resources


Common name Alternative name Linnaean name % ALA

Perilla shiso Perilla frutescens 61

Chia seed chia sage Salvia hispanica 58

Flax linseed Linum usitatissimum 55

Lingonberry Cowberry Vaccinium vitis-idaea 49

Camelina Gold-of-pleasure Camelina sativa 36

Purslane Portulaca Portulaca oleracea 35

Black raspberry Rubus occidentalis 33

Hemp Cannabis sativa 19



More Mammograms Equal More Mastectomies

But experts urge women to continue to get screened


TUESDAY, Sept. 13 (HealthDay News) — One of the goals of mammograms is detecting breast cancer early enough to avoid needing a mastectomy. But a new Norwegian study suggests that mastectomy rates climb higher as more women undergo the screening test.–Using national cancer data for more than 35,000 women aged 40 to 79 who were diagnosed with early or invasive breast cancers, Oslo researchers found a 31 percent increased risk of mastectomy in women invited to screening compared with a non-invited younger age group. The Norwegian breast cancer screening program began in 1996 in four counties, encompassing the country’s remaining 15 counties by 2004.–While scientists did not investigate why mastectomy rates climbed in screened groups, study author Pal Suhrke said the main reason is likely “cancer overdiagnosis,” or the detection and subsequent treatment of tumors that might grow very slowly and not pose much of a risk.–“Since the introduction of screening is associated with a more than 50 percent increase in breast cancer rates, some of these women are treated by mastectomy,” said Suhrke, a doctoral candidate in the pathology department at Oslo University Hospital. “For many, these results are surprising and disappointing because one might suspect that due to earlier detection of tumors, the number of women needing mastectomies would decrease.”–The study is published in the Sept. 13 online edition of BMJ. Suhrke and his colleagues found that the country’s annual mastectomy rate rose by 9 percent in women aged 50 to 69 — the group invited to screening — between the pre-screening period from 1993 to 1995 and the introduction of biennial screening from 1996 to 2004. In contrast, mastectomy rates fell by 17 percent in non-invited women aged 40 to 49 and 13 percent in non-invited women aged 70 to 79.—Dr. Stephanie Bernik, chief of surgical oncology at Lenox Hill Hospital in New York City, said the study raises some valid points, but “they haven’t addressed all the issues,” such as the idea that some breast cancer patients opt for more radical surgery because they don’t want to worry about the potential for recurrence of even early-stage cancers.–The researchers also noted that some Norwegian women in the study didn’t live near a radiation center, Bernik said, making mastectomy a safer option because follow-up radiation treatments were not otherwise accessible. Breast reconstruction techniques have also improved greatly in the past decade, she added.–“I think it’s true, if you screen more you’re going to find more cancers. That ultimately should lead to better survival for these patients,” she said. “Mastectomy is not as dreadful a choice as it used to be.”[U1] Indeed, a recent study by the Mayo Clinic indicated that few breast cancer survivors who opt for a double mastectomy as a precautionary measure regretted their decision decades later. Twenty years after surgery, 92 percent said they would make the same decision, according to preliminary findings presented at the American Society of Breast Surgeons’ annual meeting in April.—-Bernik cautioned that the take-home message of the Norwegian study isn’t that women shouldn’t get screened. While mammograms do find some early-stage cancers that may never progress, and those women go on to be treated anyway — “you can’t pick and choose which ones are going to be a problem or not . . . and so there is going to be an element of overtreatment,” she said.—-“I think we should continue screening and work toward ways of potentially trying to figure out who needs more extensive surgery,” Bernik added.

SOURCES: Pal Suhrke, doctoral candidate, department of pathology, Oslo University Hospital, Norway; Stephanie Bernik, M.D., chief, surgical oncology, Lenox Hill Hospital, New York City; Sept. 13, 2011 BMJ, online



Exposure to ‘White’ Light LEDs Appears to Suppress Body’s Production of Melatonin More Than Certain Other Lights


ScienceDaily (Sep. 12, 2011) — Exposure to the light of white LED bulbs, it turns out, suppresses melatonin 5 times more than exposure to the light of high pressure sodium bulbs that give off an orange-yellow light. “Just as there are regulations and standards for ‘classic’ pollutants, there should also be regulations and rules for the pollution stemming from artificial light at night,” says Prof. Abraham Haim of the University of Haifa.

“White” light bulbs that emit light at shorter wavelengths are greater suppressors of the body’s production of melatonin than bulbs emitting orange-yellow light, a new international study has revealed.

Melatonin is a compound that adjusts our biological clock and is known for its anti-oxidant and anti-cancerous properties.–The study investigated the influence of different types of bulbs on “light pollution” and the suppression of melatonin, with the researchers recommending several steps that should be taken to balance the need to save energy and protecting public health. –“Just as there are regulations and standards for ‘classic’ pollutants, there should also be regulations and rules for pollution stemming from artificial light at night,” says Prof. Abraham Haim, head of the Center for Interdisciplinary Chronobiological Research at the University of Haifa and the Israeli partner in the research.—The study, by Fabio Falchi, Pierantonio Cinzano, Christopher D. Elvidge, David M. Keith and Abraham Haim, was recently published in the Journal of Environmental Management. –The fact that “white” artificial light (which is actually blue light on the spectrum, emitted at wavelengths of between 440-500 nanometers) suppresses the production of melatonin in the brain’s pineal gland is already known. Also known is the fact that suppressing the production of melatonin, which is responsible, among other things, for the regulation of our biological clock, causes behavior disruptions and health problems.–In this study, conducted by astronomers, physicists and biologists from ISTIL- Light Pollution Science and Technology Institute in Italy, the National Geophysical Data Center in Boulder, Colorado, and the University of Haifa, researchers for the first time examined the differences in melatonin suppression in a various types of light bulbs, primarily those used for outdoor illumination, such as streetlights, road lighting, mall lighting and the like.–In the first, analytical part of the study, the researchers, relying on various data, calculated the wavelength and energy output of bulbs that are generally used for outdoor lighting. Next, they compared that information with existing research regarding melatonin suppression to determine the melatonin suppression level of each bulb type.–Taking into account the necessity for artificial lighting in cities, as well as the importance of energy-saving bulbs, the research team took as a reference point the level of melatonin suppression by a high-pressure sodium (HPS) bulb, a bulb that gives off orange-yellow light and is often used for street and road lighting, and compared the data from the other bulbs to that one.–From this comparison it emerged that the metal halide bulb, which gives off a white light and is used for stadium lighting, among other uses, suppresses melatonin at a rate more than 3 times greater than the HPS bulb, while the light-emitting diode (LED) bulb, which also gives off a white light, suppresses melatonin at a rate more than 5 times higher than the HPS bulb.

“The current migration from the now widely used sodium lamps to white lamps will increase melatonin suppression in humans and animals,” the researchers say.

The researchers make some concrete suggestions that could alter the situation without throwing our world into total darkness, but first and foremost, they assert that it is necessary to understand that artificial light creates “light pollution” that ought to be addressed in the realms of regulation and legislation.–Their first suggestion of course, is to limit the use of “white” light to those instances where it is absolutely necessary. Another suggestion is to adjust lampposts so that their light is not directed beyond the horizon, which would significantly reduce light pollution. They also advise against “over-lighting,” using only the amount of light needed for a task, and, of course, to simply turn off lighting when not in use — “Just like we all turn off the light when we leave the room. This is the first and primary way to save energy,” the researchers say.–“Most Italian regions have legislations to lower the impact of light pollution, but they still lack a regulation on the spectrum emitted by lamps. Unless legislation is updated soon, with the current trend toward sources as white LEDs, which emit a huge amount of blue light, we will enter a period of elevated negative effects of light at night on human health and environment. Lamp manufacturers cannot claim that they don’t know about the consequences of artificial light at night,” says Dr. Fabio Falchi of ISTIL.—“As a first step in Israel, for example, the Standards Institution of Israel should obligate bulb importers to state clearly on their packaging what wavelengths are produced by each bulb. If wavelength indeed influences melatonin production, this is information that needs to be brought to the public’s attention, so consumers can decide whether to buy this lighting or not,” Prof. Haim says. –Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Haifa.

Journal Reference-Fabio Falchi, Pierantonio Cinzano, Christopher D. Elvidge, David M. Keith, Abraham Haim. Limiting the impact of light pollution on human health, environment and stellar visibility. Journal of Environmental Management, 2011; 92 (10): 2714 DOI: 10.1016/j.jenvman.2011.06.029



Pituitary Hormone TSH Found to Directly Influence Bone Growth


ScienceDaily (Sep. 13, 2011) — Researchers at Mount Sinai School of Medicine have found that thyroid-stimulating hormone (TSH), a hormone produced in the anterior pituitary gland that regulates endocrine function in the thyroid gland, can promote bone growth independent of its usual thyroid functions. The research suggests that TSH, or drugs that mimic its affect on bone, may be key to possible future treatments for osteoporosis and other conditions involving bone loss, such as cancer. The findings were published online this week in Proceedings of the National Academy of Sciences.—The same Mount Sinai researchers had previously published research showing that TSH inhibits the creation of osteoclasts, a type of cell that removes bone tissue from the body. With this new study, however, the researchers have established for the first time that TSH also activates osteoblasts, which are cells responsible for bone formation.–“There are relatively few treatments right now for osteoporosis, and virtually all of them focus on limiting osteoclasts — that is, fighting the loss of existing bone,” said Terry F. Davies, MD, FRCP, FACE, Florence and Theodore Baumritter Professor of Medicine, Mount Sinai School of Medicine. “However, our study shows that future progress in osteoporosis therapies may hinge on medications that can mimic the effects of TSH and promote the growth of new bone. The key will be to develop TSH analogs that would activate osteoblasts and yet not affect the thyroid gland the way TSH itself does.”–“Osteoporosis is really an imbalance in the functions that create and destroy bone in the body,” said Mone Zaidi, MD, PhD, FRCP, FACE, Hon MD, Professor of Medicine, and Director of the Mount Sinai Bone Program, Mount Sinai School of Medicine. “Our findings indicate that there may be a novel new method for addressing the lack of bone production. Our discovery that TSH causes bone growth also represents a new way of thinking about the role of certain glands and how they operate.”-About 60 million people in the United States have symptoms of osteoporosis, and often they are unaware of the condition until they experience a broken bone or shrinkage of their skeleton. The disease affects women more often than men, and risk factors include aging; low body weight; low levels of the sex hormone estrogen; smoking; and some medications. Dr. Zaidi is a named inventor of a pending patent application related to the use of TSH in the inhibition of TNF activity. This patent has been filed by the Mount Sinai School of Medicine. In the event the patent is licensed, Dr. Zaidi would be entitled to a share of a any proceeds Mount Sinai School of Medicine receives from the license. Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by The Mount Sinai Hospital / Mount Sinai School of Medicine, via EurekAlert!, a service of AAAS.— Journal Reference-R. Baliram, R. Latif, J. Berkowitz, S. Frid, G. Colaianni, L. Sun, M. Zaidi, T. F. Davies. Thyroid-stimulating hormone induces a Wnt-dependent, feed-forward loop for osteoblastogenesis in embryonic stem cell cultures. Proceedings of the National Academy of Sciences, 2011; DOI: 10.1073/pnas.1110286108






[U1]WHO ARE THEY KIDDING!!!! This would be equivalent to asking them to cut off there sex organs or there limbs—“ but today that is not so bad” are they on something here!!! A loss is always traumatic especially when it is induced unnecessarily by the medical field






Show of the Week September 30 2011


Rise of Antibiotic-Resistant Gonorrhea Needs Urgent Action


Copper Reduces Infection Risk by More Than 40 Per Cent, Experts Say


Low-Fat Yogurt Intake When Pregnant Linked to Increased Risk of Child Asthma and Hay Fever


Resisting Ill health through Regulation


An Apple or Pear a Day May Keep Strokes Away





Rise of Antibiotic-Resistant Gonorrhea Needs Urgent Action

ScienceDaily (Sep. 19, 2011) — Gonorrhea is evolving into a scourge resistant to most antibiotics, and urgent action is needed to combat this public health threat, states an editorial in CMAJ.–Gonorrhea, a sexually transmitted disease (STD) that causes pelvic inflammatory disease and urethritis, is evolving into multiresistant bacteria because most treatments are now ineffective. Japan reported the first example of multiresistant gonorrhea. Neisseria gonorrhea, the organism responsible, can mutate rapidly and has a complex biology that has foiled attempts to develop a vaccine[U1]. It can also promote antibiotic resistance in other microbes through gene transfer[U2].–Targeted approaches aimed at high-risk groups, such as sexually active young people, can help stop the spread of gonorrhea. Creative and humorous public education campaigns, such as Alberta’s campaign against STDs, which resulted in a 15% to 20% increase in testing in its first month, are a start. Access to testing and care must be made easier, and emphasis on safe sex and condom use is crucial.[U3] “The spectre of widespread multiresistant gonorrhea demands an urgent public health, community and individual response,” conclude the authors. “Without action, we are heading back to the pre-antibiotic era, with an escalation in the number of deaths from other multiresistant organisms as well as rampant gonococcal infections — with treatment options for urethritis limited to painkillers, baths and catheterization for strictures. [U4]Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Canadian Medical Association Journal, via EurekAlert!, a service of AAAS.



Copper Reduces Infection Risk by More Than 40 Per Cent, Experts Say


ScienceDaily (Sep. 15, 2011) — Professor Bill Keevil, Head of the Microbiology Group and Director of the Environmental Healthcare Unit at the University of Southampton, has presented research into the mechanism by which copper exerts its antimicrobial effect on antibiotic-resistant organisms at the World Health Organization’s first International Conference on Prevention and Infection Control (ICPIC).–

‘New Insights into the Antimicrobial Mechanisms of Copper Touch Surfaces’ observes the survival of pathogens on conventional hospital touch surfaces contributes to increasing incidence and spread of antibiotic resistance and infections. Keevil proposes antimicrobial copper surfaces as one way to address this, since they achieve a rapid kill of significant bacterial, viral and fungal pathogens.—He reported studies on dry surfaces with a range of pathogens, concluding that: “Copper’s rapid destruction of pathogens could prevent mutational resistance developing and also help reduce the spread of antibiotic resistance genes to receptive and potentially more virulent organisms, as well as genes responsible for virulence. Additionally, copper touch surfaces could have a key role in preventing the transmission of healthcare-associated infections. Extensive laboratory tests have demonstrated copper’s antimicrobial efficacy against key organisms responsible for these infections, and clinical trials around the world are now reporting on its efficacy in busy, real-world environments.”The latest trial — conducted in intensive care units at three facilities in the United States — has shown that the use of antimicrobial copper surfaces in intensive care unit rooms resulted in a 40.4% reduction in the risk of acquiring a hospital infection.–

The study, funded by the US Department of Defense, was designed to determine the efficacy of antimicrobial copper in reducing the level of pathogens in hospital rooms, and whether such a reduction would translate into a lower rate of infection.—Researchers at the three hospitals involved in the trial — Memorial Sloan Kettering Cancer Center in New York, the Medical University of South Carolina (MUSC) and the Ralph H. Johnson VA Medical Center, both in Charleston, South Carolina — replaced commonly-touched items such as bed rails, overbed tray tables, nurse call buttons and IV poles with antimicrobial copper versions.–Data presented today by trial leader Dr Michael Schmidt, Professor and Vice Chairman of Microbiology and Immunology at MUSC, at ICPIC, demonstrated a 97% reduction in surface pathogens in rooms with copper surfaces, the same level achieved by “terminal” cleaning: the regimen conducted after each patient vacates a room.–Dr Schmidt said of the results: “Bacteria present on ICU room surfaces are probably responsible for 35-80% of patient infections, demonstrating how critical it is to keep hospitals clean. The copper objects used in the clinical trial supplemented cleaning protocols, lowered microbial levels, and resulted in a statistically significant reduction in the number of infections contracted by patients treated in those rooms.”Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Southampton, via AlphaGalileo.




Low-Fat Yogurt Intake When Pregnant Linked to Increased Risk of Child Asthma and Hay Fever


ScienceDaily (Sep. 18, 2011) — Eating low-fat yogurt whilst pregnant can increase the risk of your child developing asthma and allergic rhinitis (hay fever), according to recent findings.–The study will be presented at the European Respiratory Society’s (ERS) Annual Congress in Amsterdam on Sept. 25, 2011.–The study aimed to assess whether fatty acids found in dairy products could protect against the development of allergic diseases in children.–The researchers assessed milk and dairy intake during pregnancy and monitored the prevalence of asthma and allergic rhinitis using registries and questionnaires in the Danish National Birth Cohort.–The results showed that milk intake during pregnancy was not associated with increased risk of developing asthma and it actually protected against asthma development[U5]. However, women who ate low-fat yogurt with fruit once a day were 1.6-times more likely to have children who developed asthma by age 7, compared with children of women who reported no intake. They were also more likely to have allergic rhinitis and to display current asthma symptoms.—The researchers suggest that non-fat related nutrient components in the yogurt may play a part in increasing this risk. They are also looking at the possibility that low-fat yogurt intake may serve as a marker for other dietary and lifestyle factors.—Ekaterina Maslova, lead author from the Harvard School of Public Health, who has been working with data at the Centre for Fetal Programming at Statens Serum Institut, said: “This is the first study of its kind to link low-fat yogurt intake during pregnancy with an increased risk of asthma and hay fever in children. This could be due to a number of reasons and we will further investigate whether this is linked to certain nutrients or whether people who ate yogurt regularly had similar lifestyle and dietary patterns which could explain the increased risk of asthma.”

The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by European Lung Foundation, via EurekAlert!, a service of AAAS.



Resisting Ill health through Regulation


“This is a serious crisis. I need your help – and, I guess you will need mine, too. Below is the info, the proof is attached, and in this message I am making an announcement and a request. Please read this carefully – this is important to your health.” Helke

The CPSO (College of Physicians and Surgeons of Ontario) controls the licenses of some 40,000 doctors in Ontario. Because the majority of all practicing Canadian doctors are in Ontario, whatever policy this college adopts becomes basically the norm throughout Canada. Sometimes, the Maritime provinces take a different course, as they most recently did on the recognition and treatment of Lyme Disease. They also recognized MCS, at least in part, long before the rest of Canada did. Internationally, as you will see from a quick Google trip, the CPSO is respected for setting standards. The fact that under the law (Regulated Health Professions Act and Medicine Act) they are not mandated to any such thing is beside the point in this age of spin controlled by Big Pharma and corrupted medical research. Under the law, the CPSO mandated to “maintain” the evolving standards of medical care, which is fundamentally different! They act, however, as if they are in charge – and that has hurt and even killed a lot of people[U6]. Stopping the CPSO in its delusional behavior is an act of self-determination and good for your, my, and the public’s health.–In 1997, the CPSO initiated the so-called CAM Policy (Complementary and Alternative Medicine) because the public uproar and mainstream media coverage of the then running Krop case had become intolerable to their image and power. The provincial government also leaned on the CPSO on this circus of a trial[U7]. You will recall, those of you who are familiar with the case of Dr. Jozef Krop, that he was accused of diagnosing and treating MCS. In those charges, the main issues were that he told people not to use fragrances, protect themselves from toxic fumes from cleaners, cars, clothes etc. etc. and not use lawn pesticides etc. The final decision by the College in his case was a reprimand in which he was ordered to tell his patients, in a written consent form format, that all his diagnoses and treatments are only based on “his beliefs and have no scientific validity.” The fact that during his trial his expert witnesses proving the science of environmental medicine were all university professors teaching the world over, was of course ignored. Significantly also, the prosecuting CPSO did not introduce even one (!!!) published article, research item etc., to supprt their condemnation. ONLY their opinion ruled – and their opinion all the way to 2002 was that environmental medicine was a hoax.–Meanwhile, we have laws protecting us from lawn pesticides, and last week we saw a special poster in the bathrooms – – – in the CPSO building! – – – showing a canary bird wearing a gas mask; the text informed the reader that perfumes cause severe health problems and can trigger asthma; the poster requested that no fragrance be used at all, please. Now, I should add (and this will make your blood boil or make you swoon, depending on the status of your cardiovascular system or your central nervous system) that the CPSO prosecuted a doctor from Windsor (Dr. S. Kooner) for treating asthma in ways that included protection from fragrance. That case lasted a decade and ended in a restriction to his license – still in force! – which denied him the right to treat any asthma cases at all.[U8] More than 50 GPs who used to send him their asthma cases specifically protested in writing because they now had nobody to send their environmentally- triggered asthma cases to, almost all children. The CPSO declared (I was there to hear it!) that “patient outcome is irrelevant” and restricted his license. Yes, You may want to read this line again!The CAM Policy was idiotic because prejudiced against existing science and law in 1997, it was possible to manage somehow if one wished to treat people without harming them with Big Pharma products. In 2010 the CPSO commenced its “review” of that policy and came up with a new draft which they may vote on this coming November at that month’s council meeting. This new draft policy is beyond idiotic – it is outright criminal and fraudulent. It takes one’s breath away. It is also accompanied by their having persuaded the Ontario government to remove ALL those sections from the current law that protect innovative medicine and the doctor-patient relationship. Read all about it in my submissions attached here. We are back in the Dark Ages – in fact, we are now hostages to Big Pharma and doctors may as well just use a prescription pad, forget everything else – and especially even the mainstream journals which are now squarely supportive of innovation! A huge consultation charade was commenced last summer – so the CPSO would look like truly democratic regulators who “listen” to the public, seeing their mandfate under the law is to “serve the public interest”. I cannot remember when the CPSO ever (!) did that! What this consultaion process really did do is amazing and diabolically clever: the CPSO invited everybody who knows NOTHING about complementary medicine, does not practice it, or is openly hostile to it – to comment on their policy. (Go to their website and check out the policy section). To look good, I presume, they also asked some who are not likely to agree with them – such as me, of all people who has been writing about their unlawful and callous behavior for about 16 years now. They must have been surprised that it was mostly doctors who freaked right out at this proposed policy. In short: they conducted this review exactly the way they do their kangaroo trials of those doctors who run counter to their Big Pharma friends. The evidence be damned – spin reigns supreme.-To my astonishment, they invited me to comment on the new draft policy – and I did in the first round (see second attachment). That first round was not quite as criminal as this second round. Of course, as fully expected, the CPSOP did not take anything I said into account, even though whatever I said was supported not only by mainstream published literature but also by the medical groups who actually practice CAM, and by the many patient support groups. Then came the second round this year, for which by tomorrow the deadline arrives. That second round is where the criminality becomes evident: violations of chapters 2, 7 and 12 of the Charter (i.e. freedom of association, right to life and security of the person, and right to equality before the law). No policy or law in Canada may be in contravention of the Charter – that is the highest law in the land and, therefore, this policy is vulnerable to attack – and attack we all will!


It is fraudulent because many of the key scientific and legal citations from the mainstream literature in those submissions made by patient groups as well as (!) by professional medical groups were EDITED OUT before they were placed on the CPSO website, as guaranteed when this review process started. They edited out mainstream journal articles, references to official documents written or commissioned by the Ontario government, references to organizations the CPSO evidently does not like, entire sentences, and those legal items that prove the CPSO is out of control and transgressing their jurisdiction.


I have attached my two submission of 2010 and 2011, which were requested by the CPSO. The 2010 first submission is here the way I actually submitted it. It was submitted in PDF format, but the CPSO evidently thinks nothing of hacking into a PFD file and taking out what they don’t like. I suspected this, and so I intentionally wrote my submission in such a way that no matter what they chose to edited out, there was damning evidence galore to make them unhappy. Even a censored version would be bad news for them. And it was and still is! The second time round, this year, they edited out nothing, which very much surprises me. Could there be at least one person of integrity and clear thinking at the CPSO who did not want to change anything in my submission? They even kept my name in it! In 2010 they took it out.




I have had it. This is the line in the sand. The OMA Section on Complementary Medicine is now considering legal action if this policy is adopted by the CPSO. Legal advice has already been sought and we KNOW what can be done – i.e. lots of damage which is decades overdue.—To assist this process of a coming battle, I am writing a book on the CPSO with a lot of juicy details which might be out by the November EXPO – if not, by the Total Health Show in March for sure! Few people have been as focused on the CPSO as I have been because of the circa 100+ doctors with whose cases I was involved and whom they prosecuted, often ruined, and in every case traumatized; I have also been involved with many hundreds of their patients to help them find doctors elsewhere, often outside of Canada; most of the most vulnerable ones had their lives wrecked. I also have the benefit of “moles” from the CPSO with plenty of inside information which now is going public.



An Apple or Pear a Day May Keep Strokes Away


ScienceDaily (Sep. 16, 2011) — Apples and pears may keep strokes away.

That’s the conclusion of a Dutch study published in Stroke: Journal of the American Heart Association in which researchers found that eating a lot of fruits and vegetables with white flesh may protect against stroke.

While previous studies have linked high consumption of fruits and vegetables with lower stroke risk, the researchers’ prospective work is the first to examine associations of fruits and vegetable color groups with stroke.–The color of the edible portion of fruits and vegetables reflects the presence of beneficial phytochemicals such as carotenoids and flavonoids.

Researchers examined the link between fruits and vegetable color group consumption with 10-year stroke incidence in a population-based study of 20,069 adults, with an average age of 41. The participants were free of cardiovascular diseases at the start of the study and completed a 178-item food frequency questionnaire for the previous year.


Fruits and vegetables were classified in four color groups:

Green, including dark leafy vegetables, cabbages and lettuces

Orange/Yellow, which were mostly citrus fruits

Red/Purple, which were mostly red vegetables

White, of which 55 percent were apples and pears


During 10 years of follow-up, 233 strokes were documented. Green, orange/yellow and red/purple fruits and vegetables weren’t related to stroke. However, the risk of stroke incidence was 52 percent lower for people with a high intake of white fruits and vegetables compared to people with a low intake.—Each 25 gram per day increase in white fruits and vegetable consumption was associated with a 9 percent lower risk of stroke. An average apple is 120 grams.–“To prevent stroke, it may be useful to consume considerable amounts of white fruits and vegetables,” said Linda M. Oude Griep, M.Sc., lead author of the study and a postdoctoral fellow in human nutrition at Wageningen Uninversity in the Netherlands. “For example, eating one apple a day is an easy way to increase white fruits and vegetable intake.—“However, other fruits and vegetable color groups may protect against other chronic diseases. Therefore, it remains of importance to consume a lot of fruits and vegetables.”Apples and pears are high in dietary fiber and a flavonoid called quercetin. In the study, other foods in the white category were bananas, cauliflower, chicory and cucumber.

Potatoes were classified as a starch.–Previous research on the preventive health benefits of fruits and vegetables focused on the food’s unique nutritional value and characteristics, such as the edible part of the plant, color, botanical family and its ability to provide antioxidants.


U.S. federal dietary guidelines include using color to assign nutritional value. The U.S. Preventive Health Services Taskforce recommends selecting each day vegetables from five subgroups: dark green, red/orange, legume, starchy and other vegetables.–Before the results are adopted into everyday practice, the findings should be confirmed through additional research, Oude Griep said. “It may be too early for physicians to advise patients to change their dietary habits based on these initial findings,” she said.–An accompanying editorial notes that the finding should be interpreted with cauti—on because food frequency questionnaires may not be reliable.–In addition, “the observed reduction in stroke risk might further be due to a generally healthier lifestyle of individuals consuming a diet rich in fruits and vegetables,” writes Heike Wersching, M.D., M.Sc., of Institute of Epidemiology and Social Medicine at the University of Münster, in Germany.–Study co-authors are: W.M. Monique Verschuren, Ph.D.; Daan Kromhout, M.P.H., Ph.D.; Marga C. Ocké, Ph.D.; and Johanna M. Geleijnse, Ph.D. Author disclosures are on the manuscript.-Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Heart Association, via EurekAlert!, a service of AAAS. -Journal Reference-Linda M. Oude Griep, W. M. Monique Verschuren, Daan Kromhout, Marga C. Ocké, Johanna M. Geleijnse. Colors of Fruit and Vegetables and 10-Year Incidence of Stroke. Stroke, 2011; DOI: 10.1161/STROKEAHA.110.611152




[U1]GENETIC ENGINEERING—for population reduction
[U2]How does this do this?? What program is in place to achieve this transformation?? This has military written all over it ORR BILL GATES
[U3]This theme is presented now when people are in puberty and later on when they are grown this carries over—the thought should be clean sex—Hygienic Sex—most people today have neither of these practices as a result this carries on to the problems and pregnancy is another issue—if there was an actual explaination of sex and methods of hygiene this would actually curtail some of this
[U4]The report is stating the obvious and again not dealing with the real issue—Multi resistant anything has to do with environment or lack there of and if people are utilizing things that are unhealthy- un clean—GENETICALLY ALTERED –then how can we not be having this go one—these MutliResistant strains never happened til we started to play with genetics!!!
[U6]The Canadian Version of the FDA—would not surprise me if it is the FDA
[U7]This was a physician that used both allopathic and alternative healing methods and as a result of the treatments and people were recovering then the stripped him of his licence to practice allopathy so he went into herbalism
[U8]Welcome to Canada eh!!! You come to the land flowing with Death and Money